Also, to verify the gene therapy has been installed, the bacteria could produce a chemical that causes an aesthetically pleasant color change at the paper circle (similar to color antibody diagnostics), the paper circle could change the visual pattern of the aesthetically pleasing color when the gene therapy had progressed beyond the bacterial vector and was being produced at the user, person, human, or application’s genome

Think of a way or cause for people to distribute something that is less than 1/10,000 of a $

Longevity technology: a new kind of autophagy could be linked to longevity, healthspan, and wellness, if it is functional: Noting the idea that cytoplasm might have nonorganelle enzymes floating around, catalyzing things, possibly even comminuting or nonactivating some cytochemicals: These enzymes, outside the lysosome remind me of autophagy, autophagy is linked to cytorefreshment, longevity, and wellness, it could be that the genetics of having more cytoplasm enzymes outside of organelles do something like an alternate version of autophagy, refreshing and recycling physiostructures and chemicals, so looking at a variety of mammals, notably things like multicentury lifespan whales, beavers, naked mole rats, bats, tortoises, and parrots and 400 year lifespan or longer clams, kings holly, and creosote (reminded of mitochondrial apoptosis) any cytoplasm enzymes, notably proteolytic or membraneolytic, outside of organelles (like lysosome) could be quantified at mice as to longevity wellness and healthspan effects, and if beneficial made into gene therapy, drugs, or germline enhancement or optimization at humans;

Noting autophagy is linked to longevity, wellness and healthspan, and I might have read that AMPK heightens autophagy, it could be possible to engineer larger amounts of autophagy from 1) cytoterminating chemicals, proteins or peptides as drugs (possibly kind of senolytic like), also as periodically automatically activated gene therapy; an accumulation of physiologically beneficial peptides, like AEDG heightening with each sleep occurence, could, when they accumulate to a particular amount, cause the gene therapy modified tissue to produce a new circulating amount of the new autophagy chemical, causing fresh autophagy and renewal at the living human; these likely would benefit from localization to create beneficial differences in concentration and action at different cytes and tissues;

Along with heightening cytoplasmic autophagy like effect enzymes, periodic production of autophagy as algorithmically activated with a completely beneficial physiochemical that accumulates (epithalon, thymosin, likely a variety of things, possibly even beneficially heightened circulating omega 3 DHA from accumulation of DHA from endogenous production), there is also periodic, quantitatively measured as beneficial to longevity, wellness, and healthspan, triggering of mitochondria to zap cytes, causing leukocytes to absorb them.

New to me autophagy longevity technologies: Another possibility is a periodic genetic activation of the production of a non-autoimmune activating production of antigens at the surface of cytes, possibly those at the 40th percentile or less of cyte and tissue youthfulness, where the percentile is an experimentally quantified causative function at wellness and healthspan; this could possibly be quantified with mRNA transcript production’s deviation from a beneficial range (reminds me of engineering tolerance, the 2nd standard deviation at the non-preferred side of the distribution) Periodic activation of the beneficial new form of autophagy could be linked to the accumulation of a physiologically beneficial chemical (epithalon, thymosin, omega 3 DHA);

New to me autophagy with high cytotype localization technology: I perceive I read about where two different receptor proteins, when both activated, caused a cyte to be exposed to a chemotherapy drug, causing fewer well cytes to be dosed with the chemotherapy; Responding to things like: At cytes that are at less than 30th percentile of wellness, longevity and healthspan heightening physiology, the translation/transcription protein generators receive instructions to make notably larger amounts of one normal type, but comparatively tissue and cyte unusual chemical, protein, or peptide transport channel, perhaps something that occurs less than 1/400th of a cytes’ external receptors or transport channels, and at perhaps less than 1 per 200 different cytotypes and tissues, it then becomes 1/20th to 1/40th of the proportion of the cytes external transport channels and receptors enumerated amount, (twenty or ten times as populous as previously), and then an transport channel optimized autophagy causing protein, peptide, or genetic effect causes autophagy at that cyte; The concentration of the autophagy causer is 40 times greater at the 30th percentile nonwell cytes than at well, wellness, longevity and healthspan functionally active cytes; Technologically it seems possible that autophagy could be tuned to to be activated when the transport protein heightened autophagy causer is above 30 times that at an unaffected cyte;

As a another supporting technology, it is possible a protein, peptide, or peptide effluxing transporter could be part of membrane accumulating cytotransport duo; at a well, longevity, wellness, and healthspan support heightening, functional cyte the transport and efflux of a particular protein, peptide or chemical from the duo, which could be spontaneously occuring at a nonmodified organism or human, would be about the same amount, they would omit accumulating the transported protein, peptide, or chemical at the cytoplasm; that would cause any autophagy activating chemicals, proteins, peptides, or gene products, to pass through well cytes above the 30th percentile causing a minimal amount at the cytoplasm of the 30th percentile of higher cyte; When the measure of the longevity functionality, wellness, and healthspan of an unwell cyte occurred it would cause the unwell cyte to make large numbers of the harmless chemical cytotransport structure, causing it to be 40 times more prevalent at the unwell cyte; natural endogenous circulation of things that that particular transport protein transports, would reach 40 times the usual amount, causing a gene at the cyte to make an autophagy causing molecule endogenous to they cyte; another technology is to have the autophagy causing protein, peptide, or chemical that is linked to a 40 times usual amount transport protein moeity for hieghtened transport to the 1th to 30th percentile cytes, be produced at a different area of the body, perhaps from just a few gram, or even one gram area of gene therapy;

Technologies that could discern longevity, wellness, healthspan contributive and supportive cytes above the 20th percentile: mRNA, possibly gene therapy that caused presence of nonbeneficial mRNA, cytochemicals, proteins, or peptides, to cause

Normalization algorithms create higher and higher longevity, wellness, and healthspan physiochemistry, it is possible that technologies that cause the least (0- 20th percentile) longevity, wellness, and healthspan supporting cytes to be quantified as they relate to the to other cytes at the body, to be as a relation to all the cytes at the body continuously or periodically, would cause double digit % increase in baseline longevity, wellness, and healthspan annually, this could physiochemically contribute to a percentage of longevity increase, and noting the continuous improvement of 20% more longevity, wellness, and healthspan cytes annually is likely larger than an annual accumulation of nonwell cytes, that like actuarial escape velocity, things move towards wellness, longevity, and healthspan faster than they wear out; The effect of multiple 20th percentile autophagy events causes the after autophagy organism to be accumulate towards what would previously have been 90th percentile or higher cyte function as to longevity, wellness, and healthspan physiochemistry; that similarity to actuarial escape velocity, which I have also read is called longevity escape velocity, is alogrithmically related to making each iteration of cytes that support longevity, wellness, and healthspan as the new computational basis for the percentiles. A technology I read about, which might be called gene switches, makes things like AND and OR as well as other logic forms out of simultaneous gene modification effects; The technological algorithm that supports the heightening of each iteration of 20th percentile or less’ autophagy to be based on a new foundation could be based on things like: gene therapy at the entire body causes harmless unique peptides or proteins to be produced, which then utilize mechanisms that are usual to move to the exterior cytomembrane of the cyte they are produced at, This labels each cyte with a non immunoreactive cytosurface diagnostic of what it is doing and making, it is possible the diagnostic proteins that make their way to the cytosurface are actually beneficial, noting the cyte is still alive, or also possibly beneficial to neighbor cytes, the (0-20th percentile) producers of just the completely beneficial interleukins, possibly some onconeutral neutral growth protein like BDNF, or if immunotransparency and absence of immunoreaction of any kind is beneficial, perhaps slight variations on water transport proteins (aquaporins), where variations from the usual protein amino acid sequence would let diagnostic things that cause the 40 times greater amount of transport channels to cause beneficial autophagy find and have effects on cytes at less than the 20th percentile; Gene logic could also just have quantifiable things effect logic, to cause the production of mRNA that makes the 40 times higher amount of autophagy transport channels at the cytomembrane; it is also possible that gene logic can just directly make autophagy functional effects directly at any cyte where the gene logic notes 0-20th percentile longevity, wellness, and helthspan physiochemistry;